Feil Family Brain & Mind Research Institute

Publications

Found 12 results
Author [ Title(Desc)] Type Year
Filters: Author is Ishii, Makoto  [Clear All Filters]
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Ishii M, Iadecola C.  2016.  Adipocyte-derived factors in age-related dementia and their contribution to vascular and Alzheimer pathology.. Biochim Biophys Acta. 1862(5):966-74.
Ishii M, Hiller AJ, Pham L, McGuire MJ, Iadecola C, Wang G.  2019.  Amyloid-beta modulates low-threshold activated voltage-gated L-type calcium channels of arcuate neuropeptide Y neurons leading to calcium dysregulation and hypothalamic dysfunction.. J Neurosci.
Ishii M.  2019.  Apolipoprotein B as a New Link Between Cholesterol and Alzheimer Disease.. JAMA Neurol.
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Hiller AJ, Ishii M.  2018.  Disorders of Body Weight, Sleep and Circadian Rhythm as Manifestations of Hypothalamic Dysfunction in Alzheimer's Disease.. Front Cell Neurosci. 12:471.
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McGuire MJ, Ishii M.  2016.  Leptin Dysfunction and Alzheimer's Disease: Evidence from Cellular, Animal, and Human Studies.. Cell Mol Neurobiol. 36(2):203-17.
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Ishii M, Iadecola C.  2015.  Metabolic and Non-Cognitive Manifestations of Alzheimer's Disease: The Hypothalamus as Both Culprit and Target of Pathology.. Cell Metab. 22(5):761-76.
Ishii M.  2021.  Midlife dyslipidaemia as a modifiable risk factor for later-life dementia.. Lancet Healthy Longev. 2(8):e453-e454.
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Ishii M, Iadecola C.  2020.  A Novel Neurovascular Liaison Governing the Blood-Brain Barrier.. Neuron. 107(2):205-207.
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Eruysal E, Ravdin L, Kamel H, Iadecola C, Ishii M.  2019.  Plasma lipocalin-2 levels in the preclinical stage of Alzheimer's disease.. Alzheimers Dement (Amst). 11:646-653.
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Ishii M, Kamel H, Iadecola C.  2018.  Retinol Binding Protein 4 Levels Are Not Altered in Preclinical Alzheimer's Disease and Not Associated with Cognitive Decline or Incident Dementia.. J Alzheimers Dis.
Ishii M.  2016.  The Role of the Adipocyte Hormone Leptin in Alzheimer's Disease.. Keio J Med. 65(1):21.
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Ishii M, Wang G, Racchumi G, Dyke JP, Iadecola C.  2014.  Transgenic mice overexpressing amyloid precursor protein exhibit early metabolic deficits and a pathologically low leptin state associated with hypothalamic dysfunction in arcuate neuropeptide Y neurons.. J Neurosci. 34(27):9096-106.