Feil Family Brain & Mind Research Institute

Publications

Found 17 results
Author Title [ Type(Desc)] Year
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Journal Article
Koizumi K, Hattori Y, Ahn SJi, Buendia I, Ciacciarelli A, Uekawa K, Wang G, Hiller A, Zhao L, Voss HU et al..  2018.  Apoε4 disrupts neurovascular regulation and undermines white matter integrity and cognitive function.. Nat Commun. 9(1):3816.
Uekawa K, Hattori Y, Ahn SJi, Seo J, Casey N, Anfray A, Zhou P, Luo W, Anrather J, Park L et al..  2023.  Border-associated macrophages promote cerebral amyloid angiopathy and cognitive impairment through vascular oxidative stress.. Mol Neurodegener. 18(1):73.
Faraco G, Park L, Anrather J, Iadecola C.  2017.  Brain perivascular macrophages: characterization and functional roles in health and disease.. J Mol Med (Berl). 95(11):1143-1152.
Iadecola C, Anfray A, Schaeffer S, Hattori Y, Santisteban M, Casey N, Wang G, Strickland M, Zhou P, Holtzman D et al..  2023.  Cell autonomous role of border associated macrophages in ApoE4 neurovascular dysfunction and susceptibility to white matter injury.. Res Sq.
Anfray A, Schaeffer S, Hattori Y, Santisteban MM, Casey N, Wang G, Strickland M, Zhou P, Holtzman DM, Anrather J et al..  2024.  A cell-autonomous role for border-associated macrophages in ApoE4 neurovascular dysfunction and susceptibility to white matter injury.. Nat Neurosci.
Garcia-Bonilla L, Park L, Iadecola C.  2014.  Commentary on Myers et al.: growing role of the innate immunity receptor CD36 in central nervous system diseases.. Exp Neurol. 261:633-7.
Koizumi K, Wang G, Park L.  2016.  Endothelial Dysfunction and Amyloid-β-Induced Neurovascular Alterations.. Cell Mol Neurobiol. 36(2):155-65.
Platholi J, Marongiu R, Park L, Yu F, Sommer G, Weinberger R, Tower W, Milner TA, Glass MJ.  2023.  Hippocampal glial inflammatory markers are differentially altered in a novel mouse model of perimenopausal cerebral amyloid angiopathy.. Front Aging Neurosci. 15:1280218.
Alexander C, Li T, Hattori Y, Chiu D, Frost GR, Jonas L, Liu C, Anderson CJ, Wong E, Park L et al..  2022.  Hypoxia Inducible Factor-1α binds and activates γ-secretase for Aβ production under hypoxia and cerebral hypoperfusion.. Mol Psychiatry.
Santisteban MM, Schaeffer S, Anfray A, Faraco G, Brea D, Wang G, Sobanko MJ, Sciortino R, Racchumi G, Waisman A et al..  2023.  Meningeal interleukin-17-producing T cells mediate cognitive impairment in a mouse model of salt-sensitive hypertension.. Nat Neurosci.
Hernández JCCruz, Bracko O, Kersbergen CJ, Muse V, Haft-Javaherian M, Berg M, Park L, Vinarcsik LK, Ivasyk I, Rivera DA et al..  2019.  Neutrophil adhesion in brain capillaries reduces cortical blood flow and impairs memory function in Alzheimer's disease mouse models.. Nat Neurosci. 22(3):413-420.
Uekawa K, Koizumi K, Hwang J, Brunier N, Hattori Y, Zhou P, Park L.  2016.  Obligatory Role of EP1 Receptors in the Increase in Cerebral Blood Flow Produced by Hypercapnia in the Mice.. PLoS One. 11(9):e0163329.
Park L, Hochrainer K, Hattori Y, Ahn SJi, Anfray A, Wang G, Uekawa K, Seo J, Palfini V, Blanco I et al..  2020.  Tau induces PSD95-neuronal NOS uncoupling and neurovascular dysfunction independent of neurodegeneration.. Nat Neurosci. 23(9):1079-1089.
Park L, Zhou J, Koizumi K, Wang G, Anfray A, Ahn SJi, Seo J, Zhou P, Zhao L, Paul S et al..  2020.  tPA Deficiency Underlies Neurovascular Coupling Dysfunction by Amyloid-β.. J Neurosci. 40(42):8160-8173.
Uekawa K, Anfray A, Ahn SJi, Casey N, Seo J, Zhou P, Iadecola C, Park L.  2024.  tPA supplementation preserves neurovascular and cognitive function in Tg2576 mice.. Alzheimers Dement. 20(7):4572-4582.
Ruiz-Uribe NE, Bracko O, Swallow M, Omurzakov A, Dash S, Uchida H, Xiang D, Haft-Javaherian M, Falkenhain K, Lamont ME et al..  2023.  Vascular oxidative stress causes neutrophil arrest in brain capillaries, leading to decreased cerebral blood flow and contributing to memory impairment in a mouse model of Alzheimer’s disease.. bioRxiv.
Faraco G, Wijasa TStella, Park L, Moore J, Anrather J, Iadecola C.  2014.  Water deprivation induces neurovascular and cognitive dysfunction through vasopressin-induced oxidative stress.. J Cereb Blood Flow Metab. 34(5):852-60.