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A
Ishii M
,
Hiller AJ
,
Pham L
,
McGuire MJ
,
Iadecola C
,
Wang G
. 2019.
Amyloid-beta modulates low-threshold activated voltage-gated L-type calcium channels of arcuate neuropeptide Y neurons leading to calcium dysregulation and hypothalamic dysfunction.
.
J Neurosci.
Koizumi K
,
Hattori Y
,
Ahn SJi
,
Buendia I
,
Ciacciarelli A
,
Uekawa K
,
Wang G
,
Hiller A
,
Zhao L
,
Voss HU
et al.
. 2018.
Apoε4 disrupts neurovascular regulation and undermines white matter integrity and cognitive function.
.
Nat Commun. 9(1):3816.
C
Pickel VM
,
Bourie F
,
Chan J
,
Mackie K
,
Lane DA
,
Wang G
. 2019.
Chronic adolescent exposure to ∆9-tetrahydrocannabinol decreases NMDA current and extrasynaptic plasmalemmal density of NMDA GluN1 subunits in the prelimbic cortex of adult male mice.
.
Neuropsychopharmacology.
D
Faraco G
,
Brea D
,
Garcia-Bonilla L
,
Wang G
,
Racchumi G
,
Chang H
,
Buendia I
,
Santisteban MM
,
Segarra SG
,
Koizumi K
et al.
. 2018.
Dietary salt promotes neurovascular and cognitive dysfunction through a gut-initiated TH17 response.
.
Nat Neurosci.
E
Koizumi K
,
Wang G
,
Park L
. 2016.
Endothelial Dysfunction and Amyloid-β-Induced Neurovascular Alterations.
.
Cell Mol Neurobiol. 36(2):155-65.
N
Glass MJ
,
Wang G
,
Coleman CG
,
Chan J
,
Ogorodnik E
,
Van Kempen TA
,
Milner TA
,
Butler SD
,
Young CN
,
Davisson RL
et al.
. 2015.
NMDA Receptor Plasticity in the Hypothalamic Paraventricular Nucleus Contributes to the Elevated Blood Pressure Produced by Angiotensin II.
.
J Neurosci. 35(26):9558-67.
R
Marques-Lopes J
,
Tesfaye E
,
Israilov S
,
Van Kempen TA
,
Wang G
,
Glass MJ
,
Pickel VM
,
Iadecola C
,
Waters EM
,
Milner TA
. 2017.
Redistribution of NMDA Receptors in Estrogen-Receptor-β-Containing Paraventricular Hypothalamic Neurons following Slow-Pressor Angiotensin II Hypertension in Female Mice with Accelerated Ovarian Failure.
.
Neuroendocrinology. 104(3):239-256.
T
Ishii M
,
Wang G
,
Racchumi G
,
Dyke JP
,
Iadecola C
. 2014.
Transgenic mice overexpressing amyloid precursor protein exhibit early metabolic deficits and a pathologically low leptin state associated with hypothalamic dysfunction in arcuate neuropeptide Y neurons.
.
J Neurosci. 34(27):9096-106.